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Thursday, December 13, 2018

'Blood glucose regulation Essay\r'

'DISCUSSION\r\n1. cond oneness how insulin changed plasm glucose dousing over the way of the experiment. The insulin changed the plasma glucose levels by raising dramatically from fasting to the one min mark, but by hour 3 it droped again to around the same levels as fasting. The glucagon remained al roughly the same on throughout the levels except that it move a bit 1 hour after the meal.\r\n2. Explain how glucagon changed plasma glucose niggardness over the feast of the experiment.\r\nGlucagon acts on liver cells to promote breakdown of animal starch into glucose and formation of glucose from lactic acid and certain aminic acids.\r\n3. Explain what caused the change in plasma ketone niggardness over the course of the experiment.\r\nKetones changed from high during fasting to impose after eating and even after hour three because it first they were needed to generate adenosine triphosphate but droped because they were not needed as very much for fuel.\r\n4. Explain how negative feedback caused the changes in plasma insulin concentration observed during the experiment.\r\nthe level of production line glucose controls secretion of both(prenominal) glucagon and insulin via negative feedback.\r\n5. Explain how negative feedback caused the changes in plasma glucagon concentration observed during the experiment.\r\n6. The insulin/glucagon ratio changed over the course of the experiment indicating changes in glucose fund and changes in qualification to growing blood glucose concentration via glycogenolysis and gluconeogenesis. State when glucose storage capability was highest and why.\r\n7. State when glycogenolysis and gluconeogenesis were highest and why.\r\n1 hour site meal because that’s when there was the highest amount of lettuce in the blood.\r\n8. Explain how a high blood ketone level helps the physical structure conserve blood glucose.\r\nWhen the concentration of ketone bodies in the blood rises above normal the ketone bodies, mo st of which are acids, must buffered.\r\n9. Restate your predictions that were sort and build the data from your experiment that supports them. Restate your predictions that were not correct and correct them, giving the data from your experiment that supports the correction.\r\n finishing\r\n1. During exercise, epinephrine and norepinephrine are released from the adrenal gland medulla. epinephrine and norepinephrine have the same effect on plasma glucose levels as glucagon. Explain how epinephrine and norepinephrine affect plasma glucose and why this is important during exercise.\r\n epinephrin and norepinephrine will be released by the adrenal medullae in response to stress. During exercise the brain will release epinephrine and norepinephrine because it has been told that the body necessarily to adjust to the new demand that must be met. Which means that the heart rate needs to increase in order to support itself and work efficiently. 3. The symptoms of diabetes mellitus acc ommodate high plasma glucose levels and ketoacidosis (blood pH decreases referable to increasing levels of ketones). Explain how diabetes causes these symptoms.\r\nDiabetic ketoacidosis is a dangerous complication of diabetes mellitus because the chemical balance of the body becomes too acidic.\r\n'

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